ADALBERTO MOSQUEDA TAYLOR PDF
Adalberto Mosqueda-Taylor’s 2 research works with 39 reads, including: Oct-4 and CD44 in epithelial stem cells like of benign odontogenic lesions. Adalberto. Adalberto Mosqueda of Metropolitan Autonomous University, Mexico City (UAM). Toussaint-Caire S, Vega-Memíje ME, Mosqueda-Taylor A, Cuevas-González. Adalberto Mosqueda Taylor is the author of Oral Medicine And Pathology At A Glance ( avg rating, 12 ratings, 0 reviews, published ), Oral Medicin.
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Galectin-3—a jack-of-all-trades in cancer. This article has been cited by other articles in PMC.
Journal of Cranio-Maxillofacial Surgery. The clinical and pathological features of 12 AL that affected the palate are presented. The distribution of histological types and the relative frequencies of OT by centers of diagnosis are shown in Table 2.
Primordial odontogenic tumor: An immunohistochemical profile
Given the rarity of this tumor and the small number of cases reported to date, it is necessary to collect new cases in order to obtain a larger sample, towards a better understanding of the histogenesis and tumoral behavior of this condition. There were 14 female and 6 male patients. Conservative treatment including laser surgery more. The frequency of OT diagnosed at the four diagnostic centers ranged from 0.
For the statistical analysis, commercially available software SPSS Open in a separate window. Results The epithelial cells that cover the periphery of the tumor showed immunopositivity for Cytokeratins 14 and 19, while Amelogenin, Glut-1, MOC, Caveolin The highest frequency of OT 3.
Management and preservation of the soft palate is dependent on clinical stage and tumor histology. Open in a separate window. Table 2 Clinico-pathological and radiographic features of 4 cases of Primordial Odontogenic Tumour. The catalog of human cytokeratins: Odontogenic tumors derive from the tooth-producing tissues or its remnants, which are entrapped either within the jawbones or in the adjacent soft tissues.
A Mosqueda-Taylor Medicina oral, patologia oral y cirugia bucal 13 9E Their incidence seems to be higher in certain Asian and Latin American countries, where most of the reported series reveal a predominance of T-cell lineage and a Extent of resection must be tailored with tumor size. The relative frequency of odontogenic tumors in the present study was 1.
Glucose Transporter Glut-1 is found in normal cells, such as erythrocytes and endothelium at the blood-brain barrier in perineural cells, and in the basal layer of oral epithelium and epidermis.
Continuous variables were categorized to facilitate data analysis and presentation. Published online Dec 6. Reclassification of odontogenic keratocyst as tumour.
We report the results obtained after management with laser surgery and soft palate preservation in three patients with salivary gland neoplasms.
Adalberto Mosqueda-Taylor – Citas de Google Académico
The strong expression of this protein in the epithelium of POT indicates the existence of high glucose transport requirements in this pathological condition. Regulation of cancer-related gene expression by galectin-3 and the molecular mechanism of its nuclear import pathway. A total of 15, oral biopsies were registered during the year period of this retrospective study, in which we found cases diagnosed as OT.
Reduced expression of PTEN has also been reported in some oral cancers, in studies that suggest PTEN plays an important role at signaling pathways in the carcinogenesis of head and neck squamous cell carcinoma Studies of OT from several parts of the world indicate that knowing the frequency and basic clinical features of these lesions is important because they allow to know more precisely the expression of these lesions in the diverse populations, which in turn help to identify the groups at risk and possible factors associated to their development, as well as to develop more precisely differential diagnoses 2.
Support Center Support Center. Odontogenic tumors OT comprise an infrequent group of lesions arising from the tooth-producing tissues or its remnants, and these range from hamartomatous or non-neoplastic proliferations to benign and malignant neoplasm with variable aggressiveness and metastatic potential 12.
Immunophenotypic studies were performed in nine of these cases. Diagnoses were re-evaluated and the tumors classified according to the latest World Health Organization Classification of Tumors.